Filtyp
För fyra år sedan,15 oktober 1993, kom vi på idén till en personlig elektronisk assistent, Isaac, för människor med utvecklingsstörning. Inspirationskällan var bl.a. den fickdator med pekskärm, Newton, som datorföretaget Apple nyss hadesläppt. Här berättas en kortversion av denna 4-åriga sannsaga med tyngdpunkt på dess konsekvenser för brukare och forskare. Sannsagan handlar om innehåll och metod,
Developing correlations between various soil parameters are considered the major challenge to diminish time and cost associated with field measurements. Soil hydraulic and physical properties were extensively characterized using tension disc infiltrometer and soil sampling in a silty clay agricultural field in Kalaat El-Andalous, Tunisia. Tension disc infiltrometer experiments were conducted at 10
The hypothesis of this thesis is that climate models should take structural vegetation changes that operate on interannual to centennial time scales into account in order to simulate climate change in a more realistic manner. To evaluate this hypothesis, a regional Earth System Model (ESM) has been developed, validated and applied over Europe. The model has enabled the identification of a number o
Evaluation of incidence and survival of patients with borderline ovarian tumors in Sweden MATERIAL AND METHODS: All women diagnosed with borderline ovarian tumor in the Swedish Cancer Register 1960-2007 (n=6,252) combined with follow-up in the Swedish Death Registry to 1 July 2009 were included. Estimation of age-standardized relative survival rate according to time periods for diagnosis RESULTS:
Dagligen möter vi det brutala fysiska våldet. På tv-skärmar och löpsedlar. Våldet sysselsätter vår fantasi. Fascinerar, underhåller, skrämmer och säljs som mediavaror. Produkter vars konsumtion får oss att uppleva vår värld som en mer otrygg och skrämmande plats. De skapar rädsla, som i sin tur låter sig exploateras av politiskt konservativa krafter. Det medialiserade våldet och rädsleproduktionen
Spinal and bulbar muscular atrophy is a hereditary motor neuron disease caused by trinucleotide repeat expansion in the androgen receptor gene. The disease mechanism probably involves a toxic gain of function in the mutant protein, because other mutations that cause a loss of androgen receptor function result in a different phenotype and the mutant protein is toxic in mouse models. In these models
