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β decay of26Ne
Mannanase Man26A from Cellulomonas fimi has a mannan-binding module
RIFM fragrance ingredient safety assessment, 2,6-nonadienenitrile, CAS Registry Number 67019-89-0
TLR4-mediated release of heparin-binding protein in human airways : a co-stimulatory role for IL-26
The neutrophil-mobilizing cytokine interleukin-26 in the airways of long-term tobacco smokers
Long-term tobacco smokers with chronic obstructive pulmonary disease (COPD) or chronic bronchitis display an excessive accumulation of neutrophils in the airways; an inflammation that responds poorly to established therapy. Thus, there is a need to identify new molecular targets for the development of effective therapy. Here, we hypothesized that the neutrophil-mobilizing cytokine interleukin (IL)
β -delayed proton emission from P 26 and S 27
t(3;21)(q26;q22) with AML1 rearrangement in a de novo childhood acute monoblastic leukaemia
Eotaxin-3 (CCL26) exerts innate host defense activities that are modulated by mast cell proteases
BackgroundDuring bacterial infections of the airways, a Th1-profiled inflammation promotes the production of several host defense proteins and peptides with antibacterial activities including -defensins, ELR-negative CXC chemokines, and the cathelicidin LL-37. These are downregulated by Th2 cytokines of the allergic response. Instead, the eosinophil-recruiting chemokines eotaxin-1/CCL11, eotaxin-2
