Registration to AI Lund event 26 November 2025
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https://www.ai.lu.se/2025-11-26/registration - 2026-04-15
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This event is fully booked! Status message Sorry...This form is closed to new submissions. Leave this field blank
https://www.ai.lu.se/2025-11-26/registration - 2026-04-15
12 November 2026 10:30 | Supervisor training The European Centre for SI-PASS is proud to present a new format to the regular supervisor training courses designed for colleagues involved in SI-PASS programs. This training combines the in-person experience with the flexibility of online components. It provides an in-depth introduction to SI-PASS, including its structure, implementation strategies, a
Apply now!Lund University Wallenberg Centre for Molecular Medicine Research School October 2025 – June 20266 ECTSInterdisciplinary and translational lecturesTechnology workshopsCareer development sessionsAdvanced training in project design & presentationAdvanced training in scientific writingTraining in public outreachInterfaces among systemsRegenerationReplacementRepair Application deadline: Sept
https://www.wcmm.lu.se/wcmm-rs/overview-and-admission/wcmm-research-school-202526 - 2026-04-15
Welcome to the department of strategic communication The course consists of two modules:Module 1: Strategic Communication and Digital Media – Culture and Society(7,5 credits)Module 2: Contemporary Public Relations (7.5 credits)Find out more about the course:SyllabusLiteratureSchedule (TBA)
https://www.isk.lu.se/en/komc26-strategic-communication-strategiccommunication-and-public-relations-digital-media - 2026-04-15
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https://www.ai.lu.se/2026-08-26/registration - 2026-04-16
Background Anti-interleukin (IL)-4Rα monoclonal antibodies (mAb) improve lung function and decreasethe number of exacerbations in patients with COPD type (T) 2 inflammation. However, the involvement ofearly innate immune responses underlying these treatment effects is not well known. We sought tounderstand the effect and mechanisms of IL-4Rα mAb treatment on bronchial epithelial cells (BECs) fromCBackgroundAnti-IL-4Rα monoclonal antibodies (mAb) improve lung function and decrease the number of exacerbations in patients with COPD type (T) 2 inflammation. However, the involvement of early innate immune responses underlying these treatment effects is not well known. We sought to understand the effect and mechanisms of IL-4Rα mAb treatment on bronchial epithelial cells from COPD patients under