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Reflux oesophagitis in heavy drinkers : Effect of ranitidine and alginate/metoclopramide

Of 1, 400 consecutive patients applying to an outpatient clinic for treatment of alcoholism, 477 complained of upper abdominal dyspepsia. After 2 weeks of abstinence, 78 had persisting complaints; of these, 72% had oesophagitis at endoscopy. The 53 patients with oesophagitis as the sole diagnosis were randomized to treatment with either ranitidine or a combination of alginate/antacid chewing table

Influence of alcohol on morbidity after colonic surgery

Postoperative morbidity after colorectal surgery was retrospectively investigated, comparing 32 alcoholics (≥60 gm of alcohol a day) to 32 controls (<25 gm of alcohol a day). The material originated from 260 male patients and the two groups were paired with respect to operative procedure, diagnosis, age-complicating cardiopulmonary diseases, weight, and smoking habits. Postoperative morbidity was

Erythrocyte Mean Cell Volume—Correlation to Drinking Pattern in Heavy Alcoholics

ABSTRACT. Erythrocyte mean cell volume (MCV) correlates well to alcohol intake in moderate alcoholism, but only about 50% of heavy drinkers have increased MCV. To evaluate the influence of the duration and extent of a drinking episode on MCV, 64 addictive alcoholics were investigated prospectively within two weeks after a drinking period. Their daily alcohol intake was 120–480 g and the actual dri

The zinc level in alcoholics receiving disulfiram treatment

On the basis of the theory that malnutrition leads to zinc deficiency, 36 severe alcohol addicts were investigated in a large clinic for alcoholism in Copenhagen. The serum zinc levels were registered on commencement/recommencement of treatment with disulphiram and one month later. A total of 16 patients completed the entire investigation. All of the participants in the trial were without clinical

Cerebrospinal fluid neurofilament light chain differentiates behavioural variant frontotemporal dementia progressors from non-progressors

Background: Distinguishing behavioural variant frontotemporal dementia (bvFTD) from non-neurodegenerative ‘non-progressor’ mimics of frontal lobe dysfunction, can be one of the most challenging clinical dilemmas. A biomarker of neuronal injury, neurofilament light chain (NfL), could reduce misdiagnosis and delay. Methods: Cerebrospinal fluid (CSF) NfL, amyloid beta 1–42 (AB42), total and phosphory