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Differentiation of myeloid leukemic cells in vitro demonstrated by microcalorimetry : Stimulation of leukemic and remission cells by IgG-binding Fc receptors

Structural requirements for the interaction of human IgM and IgA with the human Fc alpha/mu receptor

Bacterial Modulation of Fc Effector Functions

No association of primary Sjogren's syndrome with Fc gamma receptor gene variants

Oriented immobilized anti-hIgG via F(c) fragment-imprinted PHEMA cryogel for IgG purification

Isolation and some properties of an IgG Fc-binding protein from group A streptococci type 15

Interaction of the Fc part of IgG with Lancefield extracts of hemolytic streptococci. Strain specificity and activity

IgG Fc sialylation is regulated during the germinal center reaction upon immunization with different adjuvants

Surgical outcomes in patients with haemophilia A or B receiving extended half-life recombinant factor VIII and IX Fc fusion proteins : Real-world experience in the Nordic countries

Interaction between herpes simplex type 1-induced Fc receptor and human and rabbit immunoglobulin G (IgG) domains

Herpes simplex type 1-induced Fc receptor binds to the Cgamma2-Cgamma3 interface region of IgG in the area that binds staphylococcal protein A

Identification of the site on IgG Fc for interaction with streptococci of groups A, C and G

Absence of host-cell influence on binding specificity of herpes simplex virus type 1 induced Fc receptor

Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1

Studies of protein A and herpes simplex virus-1 induced Fc gamma-binding specificities. Different binding patterns for IgG3 from Caucasian and Oriental subjects

The Functional Polymorphism 844 A>G in Fc{alpha}RI (CD89) Does Not Contribute to Systemic Sclerosis or Rheumatoid Arthritis Susceptibility.

Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation

Transfusion-related acute lung injury (TRALI) remains a leading cause of transfusionrelated deaths. In most cases, anti-leukocyte antibodies in the transfusion product trigger TRALI, but not all anti-leukocyte antibodies cause TRALI. It has been shown that the anti-major histocompatibility complex (MHC) class I antibody 34-1-2S (anti-H-2Kd) causes TRALI in BALB/c mice (MHC class I haplotype H-2Kd)

Subclass-switched anti-Spike IgG3 oligoclonal cocktails strongly enhance Fc-mediated opsonization

Fractionation of rat IgG subclasses and screening for IgG Fc-binding to bacteria

Specificity of Fc receptors induced by herpes simplex virus type 1: comparison of immunoglobulin G from different animal species