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Oriented immobilized anti-hIgG via F(c) fragment-imprinted PHEMA cryogel for IgG purification
IgG Fc sialylation is regulated during the germinal center reaction upon immunization with different adjuvants
No association of primary Sjogren's syndrome with Fc gamma receptor gene variants
Surgical outcomes in patients with haemophilia A or B receiving extended half-life recombinant factor VIII and IX Fc fusion proteins : Real-world experience in the Nordic countries
Interaction of the Fc part of IgG with Lancefield extracts of hemolytic streptococci. Strain specificity and activity
Specificity of Fc receptors induced by herpes simplex virus type 1: comparison of immunoglobulin G from different animal species
Characterization of herpes simplex virus type 1-induced Fc receptor in its interaction with rabbit immunoglobulin G (IgG)
Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1
Absence of host-cell influence on binding specificity of herpes simplex virus type 1 induced Fc receptor
Herpes simplex type 1-induced Fc receptor binds to the Cgamma2-Cgamma3 interface region of IgG in the area that binds staphylococcal protein A
Identification of the site on IgG Fc for interaction with streptococci of groups A, C and G
Interaction between herpes simplex type 1-induced Fc receptor and human and rabbit immunoglobulin G (IgG) domains
Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation
Transfusion-related acute lung injury (TRALI) remains a leading cause of transfusionrelated deaths. In most cases, anti-leukocyte antibodies in the transfusion product trigger TRALI, but not all anti-leukocyte antibodies cause TRALI. It has been shown that the anti-major histocompatibility complex (MHC) class I antibody 34-1-2S (anti-H-2Kd) causes TRALI in BALB/c mice (MHC class I haplotype H-2Kd)
Subclass-switched anti-Spike IgG3 oligoclonal cocktails strongly enhance Fc-mediated opsonization
Intraventricular Infusion of TrkB-Fc Fusion Protein Promotes Ischemia-Induced Neurogenesis in Adult Rat Dentate Gyrus.
Background and Purpose-We have previously shown that delivery of brain-derived neurotrophic factor (BDNF) through direct intrahippocampal gene transduction with a viral vector suppresses the formation of new dentate granule cells triggered by global forebrain ischemia. Here, we investigated whether inhibition of endogenous BDNF alters ischemia-induced neurogenesis in the dentate gyrus. Methods-Rat