Characterisation of behavioural and neurodegenerative changes induced by intranigral 6hydroxydopamine lesions in a mouse model of Parkinsons disease Characterisation of behavioural and neurodegenerative changes induced by intranigral 6-hydroxydopamine lesions in a mouse model of Parkinson’s disease Shane Grealish, Bengt Mattsson, Peter Draxler and Anders Björklund Wallenberg Neuroscience Center,

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Journal of Parkinson’s Disease 10 (2020) 1335–1342 DOI 10.3233/JPD-202200 IOS Press 1335 Position Paper Animal Models of Parkinson’s Disease: Are They Useful or Not? Roger A. Barkera,∗ and Anders Björklundb,∗ aDepartment of Clinical Neuroscience and WT-MRC Cambridge Stem Cell Institute, Forvie Site, Cambridge, UK bDepartment of Experimental Medical Science, Wallenberg Neuroscience Center, Lund Un

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CHAPTER 15 - Neural grafting in Parkinson’s disease: unraveling the mechanisms underlying graft-induced dyskinesia A. Bjorklund and M. A. Cenci (Eds.) Progress in Brain Research, Vol. 184 ISSN: 0079-6123 Copyright ! 2010 Elsevier B.V. All rights reserved. CHAPTER 15 Neural grafting in Parkinson’s disease: unraveling the mechanisms underlying graft-induced dyskinesia Emma L. Lane!,†, Anders Björklu

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REVIEW Cell Therapeutics in Parkinson’s Disease Olle Lindvall & Anders Björklund # The American Society for Experimental NeuroTherapeutics, Inc. 2011 Abstract The main pathology underlying motor symptoms in Parkinson’s disease (PD) is a rather selective degenera- tion of nigrostriatal dopamine (DA) neurons. Intrastriatal transplantation of immature DA neurons, which replace those neurons that have

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OP-BRAI190181 1..15 Vector-mediated L-3,4-dihydroxyphenylalanine delivery reverses motor impairments in a primate model of Parkinson’s disease Carl Rosenblad,1,2 Qin Li,3 Elsa Y. Pioli,3 Sandra Dovero,4,5 André SLM Antunes,6 Leticia Agúndez,6 Martino Bardelli,6 R. Michael Linden,6 Els Henckaerts,6Anders Björklund,2 Erwan Bezard3,4,5 and Tomas Björklund2,7 Ever since its introduction 40 years a

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awp328 482..495 BRAIN A JOURNAL OF NEUROLOGY The A9 dopamine neuron component in grafts of ventral mesencephalon is an important determinant for recovery of motor function in a rat model of Parkinson’s disease Shane Grealish,1 Marie E. Jönsson,1 Meng Li,2 Deniz Kirik,3 Anders Björklund1 and Lachlan H. Thompson1,4 1 Division of Neurobiology, Wallenberg Neuroscience Centre, Lund University, Lund,

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Neuropeptide Y and its role in CNS disease and repair Experimental Neurology 238 (2012) 265–272 Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr Review Neuropeptide Y and its role in CNS disease and repair M. Decressac a,⁎, R.A. Barker b a Wallenberg Neuroscience Center, Department of Experimental Medical Sciences, Lu

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Cardoso_et_al-2018-Journal_of_Comparative_Neurology(1).pdf R E S E A R CH AR T I C L E Target-specific forebrain projections and appropriate synaptic inputs of hESC-derived dopamine neurons grafted to the midbrain of parkinsonian rats Tiago Cardoso1,2 | Andrew F. Adler1,2 | Bengt Mattsson1,2 | Deirdre B. Hoban1,2 | Sara Nolbrant1,2 | Jenny Nelander Wahlestedt1,2 | Agnete Kirkeby1,3 | Shane Grealis

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Journal of Parkinson’s Disease 6 (2016) 57–63 DOI 10.3233/JPD-160798 IOS Press 57 Commentary Are Stem Cell-Based Therapies for Parkinson’s Disease Ready for the Clinic in 2016? Roger A. Barkera,b,∗, Malin Parmarb, Agnete Kirkebyb, Anders Björklundb, Lachlan Thompsonc and Patrik Brundind aJohn van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Forvie

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Journal of Parkinson’s Disease 7 (2017) S23–S33 DOI 10.3233/JPD-179002 IOS Press S23 Review Replacing Dopamine Neurons in Parkinson’s Disease: How did it happen? Anders Björklunda,∗ and Olle Lindvallb,∗ aDepartment of Experimental Medical Science, Wallenberg Neuroscience Center, Lund, Sweden bDepartment of Clinical Sciences, and Lund Stem Cell Center, Division of Neurology, University Hospital, L

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Invoice VAT NO: NL858213928B01 BioPartner 4, Robert Boyleweg 4, 2333 CG, Leiden, Netherlands GenScript Biotech (Netherlands) B.V. Customer NO: G0146407P0 Order NO: U8027FG070 Invoice NO: 92364337 Invoice Date: 2020.07.21 Quote NO: BILL TO SHIP TO Jenny G Johansson Lunds Universitet Box188, Lund, -, 221 00,SE - VAT No: Lunds Universitet Jenny Johansson BMC Godsmottagning D09, Lund, -, 22241,SE 46 4

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U nc or re ct ed A ut ho r P ro of Journal of Parkinson’s Disease xx (20xx) x–xx DOI 10.3233/JPD-202061 IOS Press 1 Research Report1 BDNF Overexpression Increases Striatal D3 Receptor Level at Striatal Neurons and Exacerbates D1-Receptor Agonist-Induced Dyskinesia 2 3 4 5 Simona Scheggia,1, Francesca Rossib,1, Sara Corsib, Silvia Fannib, Elisabetta Troncib, Ludovica Congiub, Romina Vargiub, Carla

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© 20 13 L an de s B io sc ie nc e. D o no t d is tri bu te . www.landesbioscience.com Autophagy 1 Autophagy 9:8, 1–3; August 2013; © 2013 Landes Bioscience AutophAgic punctum AutophAgic punctum Punctum to: Decressac M, Mattsson B, Weikop P, Lundblad M, Jakobsson J, Björklund A. TFEB-mediated autophagy rescues midbrain dopamine neurons from α-synuclein toxicity. Proc Natl Acad Sci U S A 2013; 110:E

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JW-MDS#130009 1..9 Study of the Antidyskinetic Effect of Eltoprazine in Animal Models of Levodopa-Induced Dyskinesia Erwan Bezard, PhD,1,2,3 Elisabetta Tronci, PhD,4,5 Elsa Y. Pioli, PhD,6 Qin Li, PhD,3,6 Gregory Porras, PhD,6 Anders Bj€orklund, MD,5 and Manolo Carta, PhD4,5* 1Institute for Neurodegenerative Diseases, Bordeaux University, Bordeaux, France 2Institute for Neurodegenerative Diseases,

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