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Coronary heart disease (CHD) development is associated to a combination of lifestyle and genetic factors. A number of lifestyle risk factors is well defined, while genetic factors have not yet been well determined.
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T cells have been attributed an important role in modulating repair responses following vascular injury. The aim of this study was to investigate the role of different T cell subsets in this context.
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We investigated causal effect of completed growth, measured by adult height, on coronary heart disease (CHD), stroke and cardiovascular traits, using instrumental variable (IV) Mendelian randomization meta-analysis.
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Strong and independent associations between plasma concentration of cystatin C and risk of cardiovascular disease (CVD) suggests causal involvement of cystatin C.
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It is still unclear whether carbohydrate consumption is associated with cardiovascular disease (CVD) risk. Genetic susceptibility might modify the associations between dietary intakes and disease risk.
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Sammanfattning I takt med en ökad betydelse för IT-inriktade lösningar har den svenska lagstiftningen i flera avseende uppdaterats allteftersom det har funnits behov. Urkundsförfalskningsparagrafen i 14 kap. 1 § BrB har däremot haft samma ordalydelse sedan 1948. Uppsatsen har syftat till att genom en egen utredning klarlägga ifall det föreligger ett behov av förändring även för 14 kap. 1 § BrB. Fr
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I såväl media som inom myndigheter och organisationer har det under de senaste åren i allt högre grad uppmärksammats att migrerande arbetskraft inom jordbruket/den gröna näringen eventuellt används på ett sätt som kan strida mot arbetsrättsliga och sociala regleringar.Avsikten med föreliggande studie är framförallt att sammanställa den forskning som finns avseende olika arbetslivs- och arbetsmiljö
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To examine if overall diet quality is associated with cellular and soluble biomarkers of systemic inflammation in middle-aged individuals.
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The aims of this study were to investigate the association between smoking and incident type 2 diabetes, accounting for a large number of potential confounding factors, and to explore potential effect modifiers and intermediate factors.
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Studies of the role of dietary factors in epithelial ovarian cancer (EOC) development have been limited, and no specific dietary factors have been consistently associated with EOC risk.